Dizziness In Orthopaedic Physical Therapy Practice: History And Physical Examination Page 18

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minutes of observation is required to identify periodic
Smooth Pursuit Testing
alternating nystagmus, a horizontal jerk nystagmus that
Having the patient follow a slowly moving target,
changes direction about every two minutes and that is
no faster than 20
per second, tests smooth pursuit. A
0
indicative of midline cerebellar lesions
. Spontaneous
72
marked deficit in smooth pursuit is indicative of a de-
nystagmus may also be congenital. This variant is generally
generative cerebellar process
. Small bilateral saccades in
9
horizontal; may alternate directions but not at regular
the same direction in both eyes during smooth pursuit
intervals; increases with attention, fixation, and anxiety;
testing are indicative of spinocerebellar lesions, especially
and decreases with convergence
. Pendular nystagmus
72
Friedreich’s ataxia
. Smooth pursuit testing may also
72
occurs most commonly in patients with multiple sclerosis
be positive in patients with a severe acute peripheral
and brain stem stroke
. The presence of a pendular, a
72
vestibular lesion due to superposition of an intense
vertical or torsional jerk, skew, or a periodic alternating
spontaneous nystagmus
. Abnormal findings on smooth
9
horizontal jerk spontaneous nystagmus indicates the
pursuit testing indicate the need for referral.
need for referral. In fact, any spontaneous nystagmus
requires referral with the exception of the congenital
Hearing Examination
variant noted above.
The clinician then observes for spontaneous nys-
The CN examination may indicate hearing loss. A
tagmus in eccentric positions
. Deviation of the eye
72
conductive hearing loss results from disorders in the
in the direction of the quick phase will increase the
external or middle ear; lesions in the cochlea or the
frequency and velocity of the nystagmus (Alexander’s
cochlear nerve
cause a sensorineural hearing loss. A
10
law) in patients with a unilateral peripheral vestibular
sensorineural loss is a symptom of salicylate overdose
.
10
lesion, and it may still produce a positional nystagmus
Meniere’s disease produces a sensorineural loss that is
in accommodated patients
. Detection of gaze-evoked
9
progressive over multiple episodes
. Progressive unilateral
10,19
nystagmus on lateral or upward gaze suggests a central
sensorineural hearing loss is also a typical presentation
lesion
. In fact, a gaze-evoked horizontal nystagmus
64
of patients with acoustic neuromas
. Otosclerosis can
20
implies lesions in the cerebellar flocculus and the me-
produce both a conductive and a sensorineural hearing
dial vestibular nucleus-nucleus prepositus hypoglossus
loss
. It is the authors’ experience that many elderly
10
complex, but it can also be the effect of medications,
patients complaining of dizziness present with an un-
such as hypnotics, sedatives, and anxiolytics or alcohol
diagnosed but unrelated conductive hearing loss. Even
intoxication
. Gaze-evoked nystagmus may also be the
9,72
without associated symptoms, this constitutes a reason
result of extra-ocular muscle weakness as in myasthenia
for referral to an audiologist. The presence of symptoms
gravis
. Unsustained eye movements of low frequency
72
implicating hearing loss as part of a pathology causing
and amplitude are indicative of end-point nystagmus, a
complaints of dizziness indicates the definite need for
non-pathological variant in normal subjects
. The pres-
72
medical referral.
ence of gaze-evoked nystagmus (with the exception of
end-point nystagmus) indicates the need for referral.
Weber Test
With the Weber test
, the therapist places a tuning
10
Saccadic Eye Movements
fork (256 or 512 Hz) on the top of the patient’s skull.
Having the patient look back and forth between two
With unilateral sensorineural hearing loss, the patient
targets tests saccadic eye movements. Overshooting of
will perceive the sound as coming from the normal ear.
the target (saccade overshoot dysmetria) may be observed
With a conductive disorder, the patient perceives the
in cerebellar disorders, such as Friedreich’s ataxia
.
78
sound as coming from the abnormal ear. Midline is the
Undershooting of the target or hypometria can occur
normal response for this test
. A non-midline response
67
in patients with Parkinson’s disease
. Vertical saccadic
78
indicates the need for referral.
eye movements in patients with Wallenberg syndrome
as a result of vertebrobasilar infarction may result in
Rinne Test
eye lateropulsion requiring a corrective horizontal sac-
The Rinne test
allows the therapist to distinguish
10
cade
. Uncalled-for saccades during gaze fixation on one
9
between a sensorineural and a conductive deficit in the
of the targets can occur in patients with viral cerebellar
affected ear. Normally, air conduction of the sound of
infection, paraneoplastic syndrome, and Friedreich’s
a vibrating tuning fork (256 or 512 Hz) is perceived as
ataxia
. Macrosaccadic oscillations, which are horizontal
9
louder than bone conduction. So holding the tuning fork
saccades occurring in waxing and waning bursts with
next to the external auditory canal produces a louder
200 ms saccadic intervals induced by a gaze shift, are
sound than placing the base of the tuning fork on the
indicative of midline cerebellar disease, spinocerebel-
mastoid bone in patients with normal hearing. The same
lar degenerations, and pontine lesions
. Abnormalities
72
goes in patients with sensorineural hearing loss. However,
identified during saccadic eye movement tests indicate
in patients with conductive deficits, bone conduction will
the need for referral.
appear louder on the affected side than air conduction.
Dizziness in Orthopaedic Physical Therapy Practice:
History and Physical Examination / 239

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